perianal space: surrounds the anus, continuous with the fat of the buttocks
intersphincteric space: separates the internal and external sphincters, is continuous with the perianal space,
extends cephalad into the rectal wall
ischiorectal space: lateral and posterior to the anus
deep postanal space: between the levator ani and anococcygeal ligament, connects the two ischiorectal spaces posteriorly
supralevator space: lies above the levator ani
Pathophysiology
the infection originates from an obstructed anal crypt, which communicates with the body of an anal gland
which lies in the intersphincteric plane
initial result of an infected anal gland is an intersphincteric abscess
as this abscess enlarges, it may spread in one of several directions
if the pus spreads downwards to the anal margin (the most common route), then a perianal abscess results
if the infection spreads horizontally across the external sphincter, then an ischiorectal abscess results
if the infection remains in the intersphincteric plane, then an intersphincteric abscess results
vertical spread upwards across the levator muscle results in a supralevator abscess (least common route)
an abscess that forms posterior to the anal canal may extend into one or both ischiorectal or supralevator spaces
(horseshoe abscess)
all abscesses other than a perianal abscess are termed perirectal abscesses
~50% of anorectal abscesses will result in a chronic fistula that connects the anal gland with the skin overlying
the drainage site
Clinical Manifestations
severe anal pain is the most frequent symptom
pain is constant and not associated with bowel movements
fever is common
occasionally, a patient will present with sepsis (diabetics)
a fluctuant mass is usually visible for perianal and ischiorectal abscesses
intersphincteric abscesses may not cause visible perianal skin changes, but they can be palpated on rectal exam
as a painful fluctuant mass protruding into the lumen
Supralevator Abscess
can be difficult to diagnose since some patients may have only minimal discomfort and no
external manifestations
other patients may have perianal pain and fever
a palpable mass or area of induration may be felt on rectal exam above the anal ring
may also originate from an intraabdominal source
CT scan is often necessary to make the diagnosis
Treatment
Perianal Abscess
requires incision and drainage with a cruciate-type incision
incision should be placed as close to the anal verge as possible to minimize the length of a future fistula
no need to perform immediate fistulotomy
wound packing is not necessary postop
antibiotics are usually not necessary unless the patient is febrile, diabetic, or immunocompromised
Ischiorectal Abscess
abscess cavity is often large
usually drained through a cruciate type incision
all loculations must be broken up digitally
no evidence that wound packing reduces the incidence of recurrence or fistula formation
Intersphincteric Abscess
should be drained into the anal canal by performing an internal sphincterotomy below the dentate line
abscess cavity is left open to drain
sitz baths postop will keep the wound clean
Supralevator Abscess
CT scan prior to drainage is helpful to rule out an abdominal source
should be drained internally (transanally) by incising the rectal wall overlying the abscess
should not be drained externally because a supralevator fistula may result that often requires a colostomy to heal
Horseshoe Abscess
requires draining the deep postanal space
an incision is made midway between the coccyx and anus
the anococcygeal ligament must be divided at the posterior midline to adequately drain the abscess
counter incisions are made to drain the extensions into the ischiorectal spaces
Anorectal Fistulas
Pathophysiology
occurs in up to 50% of patients with a previous perianal abscess
the fistula tract is the epithelialized communication between the internal opening in the anal canal and the
external opening through which the abscess was drained
Parks identified 4 major types of fistula that traverse the sphincter muscles: intersphincteric (45%),
transphincteric (30%), suprasphincteric (20%), extrasphincteric (5%)
in addition, a superficial fistula that doesn’t involve the sphincter muscles exists
Clinical Manifestations
patients present with persistent purulent drainage from the external opening
Diagnosis
successful treatment requires identification of the internal opening and the course of the fistula tract –
this is best done in the operating room as an exam under anesthesia
a blunt probe should be carefully passed through the external opening to identify the course of the tract
and the internal opening, taking care not to create a false passage
if the internal opening is not apparent, then hydrogen peroxide may be injected through the external opening
while looking for the bubbles in the anal canal to identify the internal opening
Goodsall’s rule can be of help when looking for the internal opening: anterior fistulas will connect to the
internal opening by a short, direct tract; posterior fistulas follow a curved tract to the posterior midline
an exception to this rule occurs for anterior fistulas that have an external opening 3 cm or more from the anal
verge - these fistulas tend to have an internal opening in the posterior midline
Initial Management
dependent on identifying the internal and external openings, the course of the track, and the amount of
sphincter muscle involved
Simple Fistulas
majority of cases (90%)
superficial fistulas, transphincteric fistulas below the anal ring, intersphincteric fistulas
Fistulotomy
contraindicated if the patient has preexisting incontinence
a probe is passed from the internal opening to the external opening, and the tract is completely opened
up and curetted
some surgeons marsupialize the tract to promote healing
cure rate is 90%
incontinence rate is minimal in properly selected patients
Complex Fistulas
cannot be treated by simple fistulotomy because of the risk of incontinence
suprasphincteric and extrasphincteric fistulas
fistulas proximal to the dentate line
any fistula involving >30% of the external sphincter
should be the first step in a patient with a complex fistula
used to encircle the tract and provide drainage of the septic process before definitive surgery
promotes fibrosis and maturation of the tract
can provide long-term control of anorectal sepsis in Crohn’s patients with multiple fistula tracts
silastic vessel loops are ideal for draining setons
Definitive Management of Complex Fistulas
Cutting Setons
used to gradually transect the fistula tract on the leading edge of the seton while allowing
scarring on the trailing edge
must divide the skin and subcutaneous tissue between the two openings
goal is to preserve sphincter continuity and preserve continence
tightened at regular intervals (every 2 weeks)
cure rates are high, but incontinence is a major risk (30%)
Advancement Flaps
used as an alternative to a cutting seton
performed in high transphincteric fistulas or persistent fistulas
internal opening is excised
a flap of mucosa, submucosa, and superficial circular muscle is created for 4 – 6 cm proximal
to the internal opening to allow for a tension-free closure
the external opening is widened and curetted
success rates reported to be 75% - 98%
Fibrin Glue
advantages include no risk of incontinence and repeatability
drainage seton is usually placed first to drain any infection
sealant is injected through the external opening into the internal opening
success rates are poor (14% - 69%)
Fistula Plug
bioprosthetic plug serves as a matrix for tissue ingrowth and to obliterate the fistula tract
plug is inserted from the internal opening to the external opening
no risk of incontinence
healing rates are < 50%
Ligation of the Intersphincteric Fistula Tract (LIFT)
ideal for transphincteric fistulas with mature tracts (prior seton drainage)
internal and external sphincters are separated in the intersphincteric space
fistula tract is identified, ligated proximally and distally, and then divided
success rates reported between 57% - 89%
Anal Fissures
Pathogenesis
results from a tear in the anoderm
90% occur in the posterior midline; 10% occur in the anterior midline
caused by local trauma, most often from a hard bowel movement
majority of patients with nonhealing fissures are found to have high anal sphincter pressures on anal manometry
– this reduces perfusion of the anoderm and impairs healing
Clinical Manifestations
tearing pain on defecation
blood on the toilet paper or stool is common
since the condition is so painful, an adequate examination may only be possible under anesthesia
anoscopy will demonstrate the fissure with the hypertrophied fibers of the internal sphincter visible at the base
associated findings include a ‘sentinel pile’ externally and an enlarged anal papilla internally
fissures in atypical locations should raise suspicions of Crohn’s disease or sexually transmitted diseases
Management
Medical Management
acute fissures will usually heal (90%) with stool softeners, bulking agents, and warm sitz baths
topical nitroglycerin or topical nifedipine have been used in chronic anal fissures to relax the internal
sphincter, which promotes healing by improving blood flow to the anal canal
medical therapy is not as efficacious as surgery, but it is not associated with the risk of incontinence
Surgical Management
reserved for patients who fail medical therapy
Lateral Internal Sphincterotomy
procedure of choice for chronic fissures that have failed medical therapy
may be done with an open or closed technique
internal sphincter is completely divided, relieving the spasm
usually done on the right side between the anterior and posterior hemorrhoidal cushions
a concomitant fissurectomy may also be performed
healing occurs in 90% to 95%
minor incontinence (leakage of mucus or gas) occurs in 10% to 20%
Botulinum Toxin Injection
relaxes the hypertonic anal sphincter, which increases blood flow and improves healing in
chronic fissures
risk of mild incontinence is low
recurrence rate is 40% - 50%
procedure may be repeated
Fissurectomy
low recurrence rate and low risk of incontinence
often combined with other procedures
Anal Advancement Flap
does not divide the internal sphincter
used in patients who are risk for incontinence after a sphincterotomy
also used to treat anal fistulas
References
Sabiston 20th ed., pgs 1402 – 1412
Schwartz, 10th ed., pgs 1225 – 1232
Cameron 11th ed., pgs 262 – 274
UpToDate. Perianal and Perirectal Abscess. Ronald Bleday, MD. Feb 24, 2020. Pgs 1 – 27.
UpToDate. Operative Management of Anorectal Fistulas. Bradley Champagne, MD, FACS, FASCRS. Dec 17, 2019. Pgs 1 – 32.
UpToDate. Anal Fissure: Surgical Management. C. Neal Ellis, MD, FACS, FASCRS, FACG. Jun 24, 2019. Pgs 1 – 20.