fractures of the 1st - 3rd ribs and scapula are often associated with other severe injuries
to the head, neck, spinal cord, lungs, and great vessels, and have a 30% mortality rate
because of associated injuries
fractures of the lower ribs (10 – 12) should increase suspicion for hepatosplenic injury
since young patients have a more flexible chest wall, rib fractures suggest a greater
transfer of force than in older patients
Diagnosis
localized pain, tenderness on palpation, or crepitus are usually present
chest x-ray or chest CT should be obtained, primarily to rule out other thoracic injuries
special rib films are not necessary
Management
pain impairs ventilation and cough, which may lead to atelectasis and pneumonia, especially
in elderly patients who may have underlying chronic lung disease
aggressive pain control without respiratory depression is the key management goal
systemic narcotics are usually sufficient for 1 – 2 rib fractures, but more extensive
injuries will benefit from epidural anesthesia or rib blocks
aggressive pulmonary toilet and chest physical therapy must also be part of the treatment
plan
Sternal Fractures
Mechanism of Injury
results from high-energy MVAs and falls
associated with other serious injuries: cardiac contusion, thoracic aortic rupture
Diagnosis
mechanism of injury, as well as sternal pain and crepitus, suggest the injury
lateral CXR or chest CT will confirm the diagnosis
Management
typically nonoperative with pulmonary toilet and analgesia
rarely open reduction and internal fixation may be required for significant chest wall
instability
if the internal mammary artery has been injured then open ligation or embolization may be
necessary
Pulmonary Injuries
Simple Pneumothorax
Pathophysiology
caused by a lung parenchymal laceration from penetrating trauma or rib fractures
air in the pleural space collapses lung tissue
ventilation/perfusion mismatch occurs when blood perfusing the nonventilated lung is not
oxygenated
if unrecognized, can evolve into a life-threatening tension pneumothorax, especially if
positive pressure ventilation is applied
Diagnosis
breath sounds are decreased on the affected side
percussion may demonstrate hyperresonance
upright chest x-ray confirms the diagnosis
Management
chest tube insertion is mandatory for a traumatic pneumothorax visible on CXR
if a patient has sustained a significant chest injury, even if a pneumothorax is not
present, a chest tube should be inserted prophylactically under the following circumstances:
requires transfer by air or ground vehicle
requires general anesthesia for treatment of other injuries
requires positive pressure ventilation
Management of Occult Pneumothorax
defined as a pneumothorax seen on CT but not on CXR
most patients can be successfully managed without chest tube placement
a small percentage of patients will have progression of the pneumothorax and require a chest
tube
development of a tension pneumothorax does not appear to be a risk
Hemothorax
Diagnosis
in the supine position, the CXR may show only a diffuse haziness
Management
any hemothorax sufficient to appear on chest x-ray should be treated with a large-bore chest
tube
a chest tube provides a way to continuously monitor ongoing blood loss and helps aid in the
decision on when to intervene operatively
in ~ 85% of cases, bleeding stops spontaneously because it originates from the low pressure
pulmonary circuit
indications for thoracotomy include initial output > 1500 mL, or persistent drainage of
200 mL/hour for 4 hours
bleeding that persists is from the systemic circulation (intercostal, internal mammary
arteries), major pulmonary veins, or heart
the pleurovac should contain an autotransfusion device so the collected blood can be given
back to the patient
if a hemothorax is not fully evacuated, an empyema or clotted hemothorax resulting in lung
entrapment can result (retained hemothorax)
Pulmonary Contusion
Pathophysiology
most common potentially lethal chest injury
often associated with a flail chest
extensive interstitial and alveolar hemorrhage leads to a ventilation/perfusion mismatch
with resulting hypoxemia
as pulmonary compliance decreases, the work of breathing increases
Diagnosis
chest x-ray shows patchy infiltrates
findings may not be initially apparent on x-ray, and may take up to 24 hours to evolve
respiratory failure may also take some hours to become manifest, and the severity of the
clinical picture may not correlate with the chest x-ray findings
Management
treatment is supportive and consists of supplemental oxygen, pain control, and pulmonary
toilet
severely hypoxic patients will require intubation and mechanical ventilation
avoid excessive volume expansion, but fluid restriction is not indicated
patients are at high risk for ventilator-associated pneumonia
contusions usually begin to resolve within 48 - 72 hours
Aortic and Cardiac Injuries
Traumatic Aortic Disruption
Mechanism of Injury
caused by abrupt deceleration injuries (high-speed, head-on collisions, falls from
significant heights)
shear forces act at points of fixation, resulting in transection
most common site is the descending aorta just distal to the left subclavian artery at the
ligamentum arteriosum, where it is fixed to the thorax
majority of patients die at the scene of free perforation and exsanguination
survivors have the rupture contained within the adventitia and tissue of the mediastinum,
forming a pseudoaneurysm
Diagnosis
specific signs and symptoms are usually absent
CXR may demonstrate a widened mediastinum, apical capping, loss of the aortic knob,
deviation of the left mainstem bronchus, left hemothorax, fracture of the 1st, 2nd ribs, and scapula
CXR alone has a high rate of missed injuries
spiral (helical) CT is the imaging test of choice based on mechanism of injury or following
up a suggestive CXR
Management
natural history is the slow expansion of the hematoma with eventual rupture
emergent bleeding should be addressed first (intra-abdominal, pelvic)
beta-blockers (esmolol) reduce aortic wall stress and should be started early, with a goal
heart rate < 80 BPM and a MAP of 60 -70 mm Hg
endovascular stent graft repair is now the treatment of choice, and may reduce the
incidence of paraplegia and mortality
when required, open surgical repair is via a left posterolateral thoracotomy
Blunt Cardiac Injury (Cardiac Contusion)
Pathophysiology
results from blunt trauma to the chest
myocardial contusions may result in arrhythmias, which are usually self-limiting
rarely, patients can present with heart failure and cardiogenic shock
most common cause of death is ventricular tachycardia or fibrillation
Risk Factors
deformed steering wheel
precordial bruises or abrasions
marked precordial tenderness
fractured sternum
bilateral rib fractures
pulmonary contusion, hemothorax
thoracic spine fractures
Diagnosis
no universally accepted diagnostic test
no correlation between troponin levels and conduction or structural abnormalities
chest x-ray is valuable for assessing pulmonary and skeletal injuries, as well as cardiac
size
EKG is required to diagnose arrhythmias
echocardiography is used to evaluate structural abnormalities in unstable patients: wall
motion abnormalities, pericardial effusions, valvular insufficiency, chordae tendineae
rupture, septal perforations, cardiac chamber rupture
Management
if one or more risk factors for blunt cardiac injury are present, then an EKG and chest
x-ray should be done
if the EKG shows an arrhythmia, then the patient should be admitted to a monitored bed for
24 – 48 hours
a cardiology consult should be obtained if the patient has a persistent arrhythmia or an
abnormal echocardiogram
may also occur in high-speed MVAs, with most patients dying at the accident scene
most injuries occur within 1-2 cm of the carina
Diagnosis
dyspnea, hemoptysis, subcutaneous emphysema, and tension pneumothorax are frequent
manifestations
chest x-ray may show pneumothorax, pneumomediastinum, and subcutaneous emphysema
massive air leak without re-expansion of the lung after chest tube insertion is highly
suggestive of a tracheobronchial injury
flexible or rigid bronchoscopy confirms the diagnosis
Management
airway control may require intubation with fiberoptic guidance
multiple chest tubes may be required to reexpand the lung
small lacerations may be treated nonoperatively if the lung can be reexpanded
larger injuries with require debridement, primary repair or segmental resection with
absorbable sutures, and coverage with an intercostal muscle flap
most tracheal injuries are best approached with a right posterolateral thoracotomy; distal
left-sided injuries must be approached with a left posterolateral thoracotomy
Esophageal Injuries
Mechanism of Injury
most commonly results from penetrating trauma with injuries to adjacent structures
mortality rate is 39%
blunt injury is extremely rare and results from forceful expulsion of gastric contents into
the esophagus from a severe blow to the upper abdomen
Diagnosis
injury is suspected by the trajectory of the weapon
chest x-ray may show pneumomediastinum, pneumothorax, or a left pleural effusion, but it may
also be normal
chest CT may reveal air adjacent to the esophagus but outside the lumen
diagnosis is made by a contrast esophagram and may be followed up with esophagoscopy in
indeterminate or negative studies
delay in diagnosis is associated with worse outcomes
Management
proximal thoracic esophagus is best approached through a right posterolateral thoracotomy
(4th or 5th interspace)
distal esophagus is approached through a left posterolateral thoracotomy (6th or 7th
interspace)
injuries diagnosed early (<24 hours) may be repaired primary, buttressed with a pleural or
intercostal muscle flap, and widely drained
leaks and fistulas are common
gastro-esophageal injuries can be reinforced with a partial fundoplication
consider a gastrostomy for decompression and a feeding jejunostomy for nutritional support
late injuries, or if mediastinitis is present, are treated with esophageal exclusion
(cervical esophagostomy, closure of the G-E junction) and wide mediastinal drainage
if inflammation is too severe, then esophagectomy may be the only option
Traversing Mediastinal Wounds
Mechanism of Injury
penetrating objects that cross the mediastinum may injure the heart, great vessels,
tracheobronchial tree, or esophagus
Diagnosis
entrance wound in one hemithorax and exit wound or missile in the contralateral hemithorax
high-risk area (‘cardiac box’) is bound by the sternal notch superiorly, costal margin
inferiorly, and the nipples laterally
Management
Hemodynamically Unstable Patient
has massive thoracic hemorrhage, tension pneumothorax, or pericardial tamponade
emergency operative thoracotomy is best approached through a left anterolateral
thoracotomy which can be extended across the sternum
Hemodynamically Stable Patient
must be thoroughly evaluated to exclude mediastinal injury
CT angiography is performed to rule out great vessel injury
esophagography and esophagoscopy are next performed to rule out esophageal injury
bronchoscopy should be performed to rule injury to the tracheobronchial tree
echocardiography is used to evaluate the heart and pericardium
may be a role for thoracoscopy in some patients
30% of patients will have a positive diagnostic evaluation requiring operative
intervention
Diaphragmatic Injuries
Diaphragmatic Rupture
Mechanism of Injury
blunt trauma produces large radial tears that lead to acute herniation; penetrating trauma
produces small defects that can take years to develop into diaphragmatic hernias
complications of acute herniation include respiratory compromise and strangulation of
peritoneal contents
left side is involved much more often than the right side – likely because the liver
protects the right side of the diaphragm
Diagnosis
commonly overlooked
chest x-ray is often misinterpreted as showing an elevated hemidiaphragm, acute gastric
dilatation, or loculated pneumothorax
the presence of bowel or stomach (N-G tube) in the thoracic cavity on chest x-ray or CT
confirms the diagnosis
thoracoscopy or laparoscopy (can miss posterior perforations) have also been used in unclear
cases
Management
should be repaired via an abdominal approach due to the high incidence of associated
abdominal injuries
most cases can be managed with debridement of nonviable tissue and primary repair of the
defect with nonabsorbable suture
a prosthetic patch is reasonable in a noncontaminated field